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The most common agent resulting in nephrotoxicity and subsequent acute tubular necrosis (ATN) in hospitalized patients is


A) contrast media.
B) antibiotics.
C) cancer chemotherapy.
D) recreational drugs.

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Which intervention has been found to retard the advancement of chronic kidney disease?


A) Calcium supplementation
B) Erythropoietin
C) Insulin
D) ACE inhibitors

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The most common cause of ischemic acute tubular necrosis (ATN) in the United States is


A) hypotension.
B) hypovolemia.
C) renal artery stenosis.
D) sepsis.

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Acute tubular necrosis can occur from (Select all that apply.)


A) increased ammonia levels from liver failure.
B) contrast dyes used for radiologic studies.
C) ischemia because of hypovolemia.
D) antibiotics that are nephrotoxic.

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The oliguric phase of acute tubular necrosis is characterized by


A) fluid excess and electrolyte imbalance.
B) fever and diminishing cognition.
C) sodium retention and potassium loss in the urine.
D) magnesium and phosphorous loss in the urine.

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The diet for a patient with chronic kidney disease (CKD) should include (Select all that apply.)


A) high carbohydrates and fats.
B) low sodium and potassium.
C) high protein.
D) low phosphorous.
E) high calorie.

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One of the most frequent causes of chronic kidney disease is


A) hypertension.
B) glomerulonephritis.
C) chronic pyelonephritis.
D) polycystic kidney disease.

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If acute tubular necrosis (ATN) does not resolve and continued tubular dysfunction ensues,the patient will then experience


A) oliguria and sodium retention.
B) infections and sepsis.
C) magnesium and phosphorus loss in urine.
D) polyuria and sodium wasting.

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The best intervention for acute kidney injury (AKI)is prevention.

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The patient most at risk for postrenal acute kidney injury is a(n)


A) elderly patient with hypertrophy of the prostate.
B) middle-aged woman with bladder infection.
C) young child with reflux at the ureterovesical junction.
D) patient who has both hypertension and diabetes.

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When a patient misses two dialysis sessions numerous electrolyte imbalances resulted.The patient will likely demonstrate (Select all that apply.)


A) lethargy because of metabolic acidosis and increased BUN.
B) skeletal muscle weakness and possible cardiac dysrhythmias because of hyperkalemia.
C) positive Chvostek and Trousseau signs because of hypomagnesemia.
D) weight gain of several pounds since her last dialysis session because of hypernatremia.
E) deep rapid breathing because of compensatory mechanism for metabolic acidosis.

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Prerenal acute kidney injury may be caused by


A) severe hypotension.
B) glomerulonephritis.
C) bilateral kidney stones.
D) acute tubular necrosis.

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The risk for contrast media-induced acute tubular necrosis (ATN) is highest in


A) a 70-year-old patient with heart failure.
B) a 50-year-old patient post gallbladder surgery.
C) a 12-year-old patient with recurrent bladder infections.
D) a 30-year-old patient with appendicitis.

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The most helpful laboratory value in monitoring the progression of declining renal function is


A) serum creatinine.
B) serum potassium.
C) blood urea nitrogen.
D) mental status changes.

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Appropriate therapy for prerenal kidney injury includes


A) fluid administration.
B) potassium supplementation.
C) fluid restriction.
D) protein restriction.

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A primary laboratory finding in end-stage chronic renal disease is


A) decreased blood urea nitrogen (BUN) .
B) decreased serum sodium.
C) metabolic alkalosis.
D) increased serum creatinine.

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Signs of late chronic renal failure include (Select all that apply.)


A) high-serum potassium levels.
B) high-serum calcium.
C) high-serum phosphorous.
D) high-blood urea nitrogen.
E) anemia.

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The condition associated with end-stage chronic renal disease that is the most immediately life threatening is


A) azotemia.
B) increased creatinine.
C) hypertension.
D) hyperkalemia.

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